[QUOTE=WhyNot]
Summary? Very, very few children have died from peanut allergies.
But, IMHO, there’s no reason for this mass hysteria and “peanut-free zones”. I’m far more worried about my kid *choking *on a peanut and dying than dying from a peanut allergy.
[/QUOTE]
Right, and thanks for the cites.
When I went to school, they served P-nut Butter sandwiches. And, they were also a common lunch item. We never heard of allergies to the extent that we do today- no school even considered “peanut free zones”.
Here is a fascinating scientific study of the peanut allergy problem in kids:
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=28527
Resolution of peanut allergy: case-control study
Here’s another from the NEJoM:
http://content.nejm.org/cgi/content/abstract/348/11/977?journalcode=nejm&minscore=5000&qbe=nejm%3BNEJMoa022613&searchid=1083134926690_136&FIRSTINDEX=0&minscore=5000&journalcode=nejm
*Conclusions Sensitization to peanut protein may occur in children through the application of peanut oil to inflamed skin. The association with soy protein could arise from cross-sensitization through common epitopes. Confirmation of these risk factors in future studies could lead to new strategies to prevent sensitization in infants who are at risk for subsequent peanut allergy. *
In other words, it seems like feeding soy milk to kids is causing them to develope peanut allergies.
P-nut allergies can be outgrown:
http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B6WH4-45SRK8K-W&_coverDate=02%2F28%2F2001&_alid=441472179&_rdoc=1&_fmt=&_orig=search&_qd=1&_cdi=6840&_sort=d&view=c&_acct=C000050221&_version=1&_urlVersion=0&_userid=10&md5=65f8150d284c92f8bd1089e5edbfd0ad
Conclusion: This study demonstrates that peanut allergy is outgrown in about 21.5% of patients. Patients with low PN-IgE levels should be offered a peanut challenge in a medical setting to demonstrate whether they can now tolerate peanuts
http://www.jacionline.org/article/PIIS009167490114683X/abstract
Volume 107, Issue 1, Pages 191-193 (January 2001)
S. Allan Bock a, Anne Muñoz-Furlong a and Hugh A. Sampson a
*Abstract
Fatal anaphylactic reactions to foods are continuing to occur, and better characterization might lead to better prevention. The objective of this report is to document the ongoing deaths and characterize these fatalities. We analyzed 32 fatal cases reported to a national registry, which was established by the American Academy of Allergy, Asthma, and Immunology, with the assistance of the Food Allergy and Anaphylaxis Network, and for which adequate data could be collected. Data were collected from multiple sources including a structured questionnaire, which was used to determine the cause of death and associated factors. The 32 individuals could be divided into 2 groups. Group 1 had sufficient data to identify peanut as the responsible food in 14 (67%) and tree nuts in 7 (33%) of cases. In group 2 subjects, 6 (55%) of the fatalities were probably due to peanut, 3 (27%) to tree nuts, and the other 2 cases were probably due to milk and fish (1 [9%] each). The sexes were equally affected; most victims were adolescents or young adults, and all but 1 subject were known to have food allergy before the fatal event. In those subjects for whom data were available, all but 1 was known to have asthma, and most of these individuals did not have epinephrine available at the time of their fatal reaction. Fatalities due to ingestion of allergenic foods in susceptible individuals remain a major health problem. In this series, peanuts and tree nuts accounted for more than 90% of the fatalities. Improved education of the profession, allergic individuals, and the public will be necessary to stop these tragedies. *
It appears that peanut allergies are really on the increase:
http://www.jacionline.org/article/PIIS009167490201429X/abstract
Abstract
Background: Allergy to peanut is common. However, it is not known whether the prevalence of sensitization and clinical allergy to peanut is increasing. Objective: We sought to determine any change in the prevalence of peanut sensitization and reactivity in early childhood in 2 sequential cohorts in the same geographic area 6 years apart. Methods: Of 2878 children born between September 1, 1994, and August 31, 1996, living on the Isle of Wight, 1273 completed questionnaires, and 1246 had skin prick tests at the age of 3 to 4 years. Those with positive skin prick test responses to peanut were subjected to oral peanut challenges, unless there was a history of immediate systemic reaction. These data were compared with information on sensitization and clinical allergy to peanut available from a previous cohort born in 1989 in the same geographic area. Results: There was a 2-fold increase in reported peanut allergy (0.5 % [6/1218] to 1.0 % [13/1273]), but the difference was nonsignificant (P = .2). Peanut sensitization increased 3-fold, with 41 (3.3 %) of 1246 children sensitized in 1994 to 1996 compared with 11 (1.1 %) of 981 sensitized 6 years ago (P = .001). Of 41 sensitized children in the current study, 10 reported a convincing clinical reaction to peanut, and 8 had positive oral challenge results, giving an overall estimate of peanut allergy of 1.5% (18/1246). Conclusions: Sensitization to peanut had increased between 1989 and 1994 to 1996. There was a strong but statistically nonsignificant trend for increase in reported peanut allergy
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed&cmd=Retrieve&list_uids=11113829&Dopt=Citation
CONCLUSIONS: The majority of children with clinical peanut hypersensitivity followed up for 5 years will have adverse reactions from accidental peanut exposure. Symptoms experienced during subsequent adverse peanut reactions may not be consistent with symptoms reported during initial reactions. Therefore proper education regarding peanut avoidance and treatment of adverse reactions is necessary in all cases of clinical peanut hypersensitivity. Young children who are allergic to peanuts can lose clinical hypersensitivity.
So, indeed, peanut allergies are getting more common, and they can be serious.