Yes; as a rule, most manufacturers (both name brand and private label) tend to use the same color schemes for their products, likely to reduce confusion and help consumers find the sweetener variety that they prefer:
Sugar is big business. There is lots of documentation on how this industry tried to smear competitive artificial sweeteners and fund research to this end. One method was by manipulating the “generally regarded as safe” category. Testing on sugar only used doses similar to what one might reasonably consume. Testing for cyclamates and other sweeteners typically used far, far higher doses (any cancer risk is considered under GRAS, which does not even test many additives used) and even then most studies did not suggest much risk in people, which are bigger than mice.
But some people consume a lot of artificially sweetened products. It is obviously worth studying this, and it has been studied up the yin yang. Rightfully so, if there are real risks identifying them is important. Most studies show little risk at reasonable doses. The study quoted in the original post, worse than most, suggests a relative risk of 1.13
If you smoke, your chance of getting lung cancer goes up from seven to twenty-five times (depending on the details). This is a huge effect and has been repeatedly shown. This is a relative risk as high as 25. Saying something increases risk is not always helpful: living in a city increases risks. Taking something rare and slightly increasing the risk still leaves something rare. Eating sweetener is not rare, but some of the claimed effects are. In some cases, relative risks under 1.3 are not significant enough to worry about, even if it genuinely reflects an increased risk, this is tiny.
Who eats sweeteners? People concerned about their weight. Is this population more likely to be obese or diabetic or have other issues? I don’t know. But nutrition studies, scientifically speaking, suck. They generally rely on surveys and imperfect memories and trusting people to use or not use things. They often compare two very similar things (a low fat diet with 30% calories from fat may not differ much from a high fat diet at 38%). And proving causation is very hard to do. Most nutrition studies are garbage and so is the way their results are reported in the media.
Still, it is the WHO. Should this give it gravitas? Only a little. It is likely true that it is better to consume less sugar and less sweetener. We know excessive sugar consumption has a number of possible detrimental effects and so should be enjoyed in moderation. These risks seem pretty tiny and barely reach statistical significance. But if you eat a lot of sweet foods it makes sense to reduce sweeteners too if you wish to. I consume sweeteners, and try to enjoy sugary foods around times I am physically active. I think sweeteners are sometimes healthier than fructose which forms 50% of table sugar (the amounts of fructose in fruit are low, easily handled by your body, and eating whole fruit is still wise).
Amen, brother! Or, as I was planning to put it: The nice thing about artificial sweeteners is that they have a useful feature that prevents me from experiencing any of their potentially harmful effects, which is that they taste like shit. Inevitably, I see a new one come along for which there is a claim for no aftertaste and I taste it and say, “Like hell there isn’t!”
And…for that matter…artificial salt (i.e., potassium chloride in place of sodium chloride) is another one of those nasty-tasting things.
I wish products that had any different sweetener or salt had to say so prominently on the label. Instead, they sucker you in with “reduced sugar” or “reduced sodium” and don’t bother to tell you that they just replaced it with something that tastes awful.
I don’t understand people’s dislike of the taste of artificial sweeteners, but I also am sure most people can’t grok the fact that normal sugar tastes “dirty” (as in, literally tastes somewhat like dirt) to me.
There actually is a non-zero risk to people with phenylketouria, hence the warning label on all aspartame products in the US, but people with that disorder are routinely identified shortly after birth, and if they aren’t it will become apparent that something is amiss before too long. But that’s a small minority of people, for most folks aspartame is a negligible risk at most.
If I am remembering the right study it was a compilation of data from multiple sources and was regarded as a low data quality review. I still drink my diet colas. Gotta die of something. Probably won’t be aspartame…
I was bummed to hear this news. When I was doing keto, sometimes I just needed a fix of something sweet-tasting, and erythritol was the most natural-tasting fake sugar in baked goods. I’d make some awesome sugar-free brownies with it, and homemade sugar-free ice cream made with almond milk, cocoa powder and erythritol was delicious; as good or better than the real thing.
Mrs. solost got me a big Costco box of monk fruit / stevia sweetener packets to put in my coffee. That’s still ok, right? Wrong! What do they use as a bulking agent to make enough fake sugar to fill a packet? That’s right, erythritol. But you don’t know that unless you read the ingredient list.
Normally I don’t worry too much about all these various health warnings, but there’s a gene that runs in our family that can cause dangerous blood clots. My sister found out she had the gene when she had pregnancy complications, which means I may have it; I’ve never been tested for it.
To me, at least, the artificial sweeteners have significantly different tastes. I prefer Sweet’N Low, I tolerate Equal, and I don’t like most of the others.
I doubt this is what you were reading, but the effect looks real
That’s a pretty good set of data. A real observation of increased frequency of events, followed by a testable theory of causation, followed by observing the events that could lead to increased clotting risk in healthy volunteers who ate amounts of erythritol that are actually found in food.
The stuff was first approved for use in food in 1990, in Japan. I’m having trouble finding how rapidly its use caught on, and how much real human experience we have of it.
Here’s the thing though: people do drop like flies and determining if a specific input, be it nutritional or behavioral, is a significant factor is very often not so obvious unless it is carefully studied
For kicks I looked at the actual erythritol study (not Nutasweet). If I used the stuff it would give me pause. These are numbers that are clinically meaningful. Solid study to my read.
I’m not saying that all sweeteners act the same, are perfectly safe for every person in any quantity, or that studying them is unimportant. I don’t knowingly eat erythritol myself.
But I am saying PubMed shows over 70,000 papers about artificial sweeteners. In addition, billions of people have consumed them daily for over five decades. It is important to know if they are safe and worth checking risk. It is also worth keeping in mind studies showing negative results are often not reported, that increased risk is not the same as significant risk and that similar levels of risk for thrombosis have been shown by having a non-O blood type.
I’m not saying it is an unimportant effect. But it needs more study and is not worth losing sleep over if lacking other risk factors. That said, I have no plans to start eating it and alternatives may be safer.
But these levels are clinically significant levels of risk. Blood type is also a significant risk (weird that non-O tends to lower BP and higher thrombosis risk …) but we don’t have any choice about that. Consuming any or any specific added sweeteners is OTOH a modifiable choice. If I could choose to be blood type O I would, lower mortality risk overall apparently.
Oh, I agree. I think there are smarter alternatives to erythritol and so these risks are avoidable. I was more speaking in general about newspaper articles saying “X increases the chance of nastiness” when this increase might be large, small or insignificant. They also do not generally say the prevalence of whatever nastiness they are trying to call attention to/stoke fear about.
So therefore the other 45% of us with a non-modifiable increased risk factor of clinical significance should be especially aware of avoiding increasing our risks significantly further?
It is good to minimize avoidable risks. I don’t knowingly use erythritol and am not going to start - there are better alternatives based on current information. But it is also worth keeping risks in perspective. (I am also not knowingly pregnant.)
(Most people do not know their Factor V Leiden status or antithrombin III levels. I don’t think most people need to get these blood tests though. More data is not always better. And we don’t likely know every risk.)