This topic never fails to piss-me off. How is it that it manages to generate so many fundamental mis-readings all at once. And somehow I’ve been at least obliquely referred to as a “totalitarian” and compared to holocaust deniers by posters who evidently can not be bothered to check the facts and fairly extensive amount of literature previously cited. I find that last part fundamentally annoying.
So then,
My Dear ** Zarathustra: **
I fail to see the * relevance * of your “newspeak” citation. As always, I am referring to the usage of the term race in a biological context, which I think should have been clear from the context of the message you responded to, * as I clearly stated the same in the self same message*. Since you have not done your homework, you’re having trouble following this. My apologies, but I do suggest if you follow through with the links you will find a full and complete argumentation. Or you can continue to argue against straw men and flail against political bogeymen.
I might add I ** was not ** arguing the term race should be discarded because ** it was politically inconvenient **, indeed I do not believe I mentioned anything political at all. If I may be so bold, I would like to point out you are the one bringing in politics with bad analogies to Trotskyites and irrelevant citations to Orwell. Perhaps if you step outside of politics and engage the science you will learn something. Otherwise, I call this merely illogical smearing based on a fundamental misreading of the comments. Deliberate? I don’t know. However, I don’t appreciate it. ** Your ** abusive mis-readings give ** me ** pause.
Autumn Wind Chick
Really now? “Race deniers” modus operendi resembles holocaust deniers?
What pray tell does this mean? My entire position is based on peer-reviewed literature in that wonderful new field, molecular genetics. Of course there is other evidence too, but the most relevant body of the evidence is there.
Now I hope I can be forgiven for taking exception to be inaccurately placed in the category of holocaust deniers – a rather disgusting and above all * inaccurate * insult. I divine from your comments that you simply do not understand the evidence. May I suggest some biology classes? Or do you have some substantive evidence to bring to the table — do recall we have dealt with this before. I would like to know what over-arching evidence for race being a useful biological category you have to demonstrate what I am missing.
JillGat
Just in regards to this conversation, you might enjoy the fairly extensive (scientific) literature citations in the linked discussions, presuming you may not have followed this before. I am not sure how to understand your observation on phenotypes or what importance you’re attaching to that, however I hope you do find the literature useful.
But, London:
Usage of race per se should not be construed as an error. Building on Jillgat’s own statement, insofar as a racial category in any given country may be useful to describe an population grouping with common socio-economic and environmental characteristics, it could very well be useful. The problem arises when observed differences are ascribed, unsupported now by the genetic evidence, to inherent biological differences between the “racial” population and whatever population it’s being contrasted with, as opposed to investigating socio-economic, environmental or cultural practices which may be inducing the difference. That’s good research design. Bad research design is the assuming of the genetic differences contra the evidence.
For example, as I recall, African Americans have elevated instances of hypertension. An error would be to ** assume, ** given present data, that this is due to some shared genetic trait through the population in contrast with say “whites.” One would be well-advised to look at social factors (impact of racial tensions, e.g., low social status ascribed), cultural factors (diet, perhaps parenting practices, etc.) among other things. And of course not assume that the Af. Am population is either genetically homogenous or homogenous with African populations. As I recall, research in this area — I’m afraid I’m operating on memory here so all caveats apply — indicated that West African populations (I believe if I recall correctly they were specifically Nigerian, so even the extrapolation to West African may be a bit much) do not, contrary to the researchers initial expectations (based on race) reflect elevated hypertension, but children of the same raised in the USA did. Interesting, no?
One might further note that the problematic of racial categorization is reflected even in the proposition that say, for example, Af Am pops did express at elevated levels a trait leading to increased risk of hypertension. As we know from other data, say sickle cell, that trait is not likely to be private to recent African descended populations. Let’s presume that it, like sickle cell, also expresses in elevated levels among Lebanese, who are ordinarily classified as “white” in popular schema and those levels are more or less analogous to the rates expressed among African Americans. What kind of observation do we make here. Quite clearly the “black” versus “white” category doesn’t work well at all, above all for truly capturing the epidemiological risks. Implicitly when the black versus white category is used, its really saying “North West European” versus “Black” — which may work for a number of risk areas, although certainly not all. However, it does distort our understanding of the issue and we do end up with analytical errors (perhaps ultimately trivial in this case given the numbers involved for a North American population) resulting from a fallacy of composition: (inappropriately lumping all blacks(*) into a risk group, inappropriately excluding some whites(**) from a risk group.) As a fast and ready rule of thumb it may be permissible, in fact useful so long as the medical practitioner understands this. For truly understanding population issues, I argue it is not and when medical practitioners do not understand this, we have, IMHO, a potentially serious problem of mis-delivery of services. I should let Edwino comment on this as we now get into his field.
(*: e.g. presuming my fictional trait has characteristics like sickle cell and not all “black African” let alone other “negroid” phenotypes express the trait)
(**: similarly, many white groups, e.g. Mediterraneans, Middle Easterners etc. may be in appropriately excluded from the risk group and thus go undiagnosed, again resulting in poor medical service delivery.)
Opus
Well-known is of course relative. I am sure lots of names well-known to me are not to you for the very good reason its not your field of interest. If you search on Rushton and my username you will find that in the past the bona fides have been provided. Reference to his falsification of data as well as cites to refutations. In re the original article in question, I do not have the particular journal available to me, however prior experience with Rushton’s work do not lead to confidence. Although I see DDG has provided some materials. I might add that his attacks on Gould are particularly disappointing since Gould at the very least is well-trained and well-respected in biological sciences, whereas he… is not. Frankly, Rushton’s charaterizations of Gould’s writing depend on his fundamental misreadings. But then his grasp of evolutionary biology is slim at best.
Oh yes, Opus, insofar as Rushton’s theories are based on (a) the presupposition of large genetic differences between “races” (b) poorly theorized and even more poorly supported a priori assumptions about supposed connexions between race based differences in (i) sex (ii) intelligence (iii) penis size (iv) climate -hot/cold- which he assumes ** a priori ** they barely merit direct refutation. However, in the context of what modern molecular genetics and its derived population information has taught us, (a) is false on its face and (b.i,ii,iii,iv) are quite simply fundamentally misconceived. You won’t find much in the way of direct refutations of him since he’s more or less in the league of “creation scientists” — real researchers have better things to do.
(Let me add parenthetically that Rushton does put whites at the top of the racial food-chain for his overall argumentation is that “whites” are “just right” — not too intellectual like Asians (in his schemata, really just the cold-calculating inscrutable Asian stereotype restates) nor too physical/emotional like blacks. Think of the bowls of porridge… Idiotic.)
Now, in re race usage: please see the past conversations, your misunderstanding about “splits” — based on a 1988 paper mind you — have already been dealt with. The genetic evidence clearly show no formal splits based on race occurred as genetic flow always remained relatively high. This has been developed in past discussions, you would do well to read them and avoid the obvious factual errors.
And in regards to suppression of “race based” differences: well if one is willing to believe in conspiracy theory, fine, but I suggest to you the body of data from modern population genetics adequately refutes the concept of “suppression” of data due to biases: You will note that no author dismisses population differences, only races as too incoherent. This has ** nothing to fuck to do with race being icky etc ad nauseum ** its about ** genetics ** and what it teaches. I hope this will finally become clear. I think when one begins to grapple with what current population genetic science is actually saying it will seem less threatening or strange.