See here for example.
GIGOBuster’s assertion that the differences created among SIRE groups by evolution is trivial is unsupported by anything except a hope that it’s trivial. Measurements contine to show profound differences among SIRE groups and most modern efforts are directed at showing the differences are environmental. But the differences themselves are profound.
It is not really just my assertion, so stop denying that most researchers know that people like you are roaming around. (And stop denying also that researchers are even looking at the reasons why guys like you are getting it wrong) And once again, it is not trivial in some aspects, but in many medical ones it is, and the big one: the difference in intelligence among races, that drives scientific racists, has virtually no evidence in the genetic field. Some people just love to confuse research (Like the one CP posted earlier) that even removes the racial aspect as being supportive for their say so’s when it is really not doing that.
But, that’s all about alleles and stuff. I don’t see them identifying any phenotypical changes we didn’t already know about.
We dont know the exact genetic relationship for thousands of components of human physiology and function. When we do measure something it frequently varies by SIRE group. For example, you would find measurable and significant differences for testosterone or creatine kinase reference levels for adult male blacks and whites. Differences in sodium handling. Differences in lactase persistence. Differences in the frequency of genes for disease states. On and on. We are markedly different among current SIRE groups genetically, and there is a reasonable inference that we are therefore markedly different from L0 200 kya.
Evolution carries on. It hasn`t stopped and there is good evidence for a marked increase over the last 40k years.
Are these changes significant? One of the best points of evidence they are significantly advantageous is the marked penetration for a given allele change in descendant populations from the point of change. MCPH1 has 70% penetration for the haplogroup D variant. We may not know the phenotypic result of that variation but it is a reasonable conclusion that a significant reproductive advantage derives from it since the penetration is so pronounced.