In simplified terms, yes.
The broad consensus (look at the diagram in your own most recent cite from Ms Brahic) is that out of africa occurred in a roughly one-way direction about 65 kya. Minimal contact between sub-saharan and non-africa lines for about 60,000 years. Non-african pools acquire Neanderthal introgression for most of their lineages because of when it occurred (near the out of africa migration, and before most subsequent splits).
Per Reich, at least, semitic genetic pools back to east africa 3kya; some subset of that into southern africa 1.5kya.
But you can go read about all this for yourself, if it’s news to you.
It’s why the out of africa gate creates two broad pools. There are plenty of interesting exceptions and nuances, which is why the idea of calling a given genetic pool a “race” doesn’t make much sense. There is plenty of diversity in both these pools, which is why trying to divide by quantity of diversity makes no sense at all.
What there is not, is passing of genetic information on any scale large enough to homogenize between the non-african, and sub-saharan pools. So introgressed gene variants (Neanderthal and Denisovan, e.g.) and ordinary new SNP variants that are selected for by evolution (1.6% of the Perelegen SNP database representing 1,800 genes across human physiology, per Eric Wang) remain for the most part of each side of that Red Sea-Sahara gate, passing only to descendant non-african or subs-saharan lines, depending on which pool got the gene variants. And within those two broad pools, how penetrated the variant is will depend on how early it showed up, and how reproductively advantageous it was (or, in some circumstances, whether it was in a founder group or present at a migration bottleneck).
Neanderthal genes are an example. Average frequency for gene variants in prostate cancer are an example. MCPH1 haplogroup D is an example. Genes driving bone density, creatine kinase levels, and plenty of others are more examples.
It isn’t that there is a perfect division of gene variants. It’s that there’s enough of an average frequency difference for gene variants that drive phenotypic outcomes to create two different pools.
Of course, there’s always the confounder of nurturing influences. Why, I was noticing just yesterday at one of the college basketball games that blacks get nurturing for basketball, and whites get nurturing for watching it. I was sad that all those young kids–overwhelmingly european–in the rows right behind their basketball stars were too lazy to go actually become those stars. Or maybe they’d rather study in their off hours than become basketball stars.
Seriously, though, I really encourage you to read the original papers and pay less attention to what is massaged into a headline for consumption by the masses. I think you’ll find the statement I’ve quoted from you above is roughly the consensus…(east africa and the sub-saharan Sahel being the exceptions and not the rule).