I hesitate to engage in conversation on this subject, sir or madam, because I suspect that your motives are somewhat distasteful and might lead me to being impolite. Well, that’s only half the reason; there’s also the fact that I’m really, really lazy. Anywho, someone in another thread has posted something that not only goes to my point but does so more competently than I could have, though with fewer wisecracks.
Would you consider taking a college-level molecular biology class to further your knowledge in this subject? I think you’ll find the subject enlightening. Here’s a bit of foreshadowing. Read carefully, I get paid to teach this stuff, there’ll be a test at the end: Human haplogroups consist of mostly silent mutations which means there’s a change in a nucleotide but due to the degeneracy (e.g. or, in layman’s terms: redundancy) of the genetic code, there’s no change in the resultant protein (the physical expression of the gene, or the phenotype).
Now, using your example, this means while Europeans and Blacks have a change in nucleotide sequence, there is no change in the final protein. I’ll repeat: there’s no change in the final protein. There are evolutionary examples in which groups carry real, phenotypic changes that are passed down (e.g. Southern Europeans & CCR5 and Sub-saharan Africans & Hemoglobin S) but these were due to infection-induced bottlenecks, and these should be considered the exceptions not the rule.
What you and the peanut gallery fail to see (because your knowledge of genes does not extend past year 1999) is that a phenotype does not emerge due to a genes alone; indeed, traits are an intricate interplay between genes and epigenetics (environment). If you need any proof of this unassailable fact, just look here where the fur coloration of a cat is different from its clone. Further, why do you think that nearly all suicide victims show epigenetic silencing of BDNF? It’s because the genes are responsive to the environment.cline not a race.